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<!--
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||
UID=105424
|
||
ConceptID=C0497406
|
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-->
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||
<!--imgCountBooks = 0--><h1 class="medgenTitle"><div class="MedGenTitleText">Overweight</div></h1><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>105424</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C0497406</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Finding</dd></dl></div></div><table class="medgenTable"><tbody><tr><td>Synonym:</td>
|
||
<td>Patient overweight</td></tr>
|
||
<tr><td><span class="bold">SNOMED CT: </span></td>
|
||
<td>Overweight (238131007); Patient overweight (238131007)</td></tr>
|
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<tr><td colspan="2" class="small"> </td></tr><tr><td>HPO:</td>
|
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<td><a target="_blank" title="Human Phenotype Ontology" href="https://hpo.jax.org/app/browse/term/HP:0025502">HP:0025502</a></td></tr>
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<div class="portlet mgSection" id="ID_100">
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<div class="portlet_head mgSectionHead ui-widget-header"><h1 class="nl" id="Definition">Definition</h1><a sid="100" href="#" class="portlet_shutter" title="Show/hide content"></a></div>
|
||
<div class="portlet_content ln">Increased body weight with a body mass index of 25-29.9 kg per square meter. [from <a title="Human Phenotype Ontology" href="http://www.human-phenotype-ontology.org" class="defSource" target="_blank">HPO</a>]</div>
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<div class="portlet mgSection" id="ID_118">
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<div class="portlet_head mgSectionHead ui-widget-header"><h1 class="nl" id="Term_Hierarchy">Term Hierarchy</h1><a sid="118" href="#" class="portlet_shutter" title="Show/hide content"></a></div>
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<div class="portlet_content ln HierarchyGTR"><div class="jig-ncbitabs"><ul><li><a href="#tabGTR">GTR</a></li><li><a href="#tabMGEN">MeSH</a></li></ul><div id="tabGTR"><div class="search_result"><div class="rprts"><div class="chiclet_legend"><span class="chiclet_list" style="position:static;"><span title="Clinical test" class="chiclet Ccolor round">C</span><span>Clinical test, </span><span title="Research test" class="chiclet Rcolor round">R</span><span>Research test, </span><span title="OMIM" class="chiclet Ocolor ">O</span><span>OMIM, </span><span title="GeneReview" class="chiclet Gcolor">G</span><span><em>GeneReviews</em>, </span><span title="ClinVar" class="chiclet Vcolor">V</span><span>ClinVar </span></span></div><div id="hierarchy" class="margin_t1"><div class="ds_tree"><ul><li class="matched_ds"><span class="TLline">Overweight</span></li></ul></div></div></div></div></div><div id="tabMGEN"><div class="ds_tree"><ul><li><span class="TLline"><a href="/medgen/867443" ref="tree=MeSH" title="MedGen record for Phenotypic abnormality">Phenotypic abnormality</a></span><ul><li><span class="TLline"><a href="/medgen/808205" ref="tree=MeSH" title="MedGen record for Growth abnormality">Growth abnormality</a></span><ul><li><span class="TLline"><a href="/medgen/892367" ref="tree=MeSH" title="MedGen record for Abnormality of body weight">Abnormality of body weight</a></span><ul><li><span class="TLline"><a href="/medgen/12145" ref="tree=MeSH" title="MedGen record for Increased body weight">Increased body weight</a></span><ul><li><span class="matched_ds">Overweight</span><ul><li><span class="TLline"><a href="/medgen/18127" ref="tree=MeSH" title="MedGen record for Obesity">Obesity</a></span><ul><li><span class="TLline"><a href="/medgen/90229" ref="tree=MeSH" title="MedGen record for Abdominal obesity">Abdominal obesity</a></span></li><li><span class="TLline"><a href="/medgen/704385" ref="tree=MeSH" title="MedGen record for Alimentary obesity">Alimentary obesity</a></span></li><li><span class="TLline"><a href="/medgen/453266" ref="tree=MeSH" title="MedGen record for Childhood obesity">Childhood obesity</a></span></li><li><span class="TLline"><a href="/medgen/1389124" ref="tree=MeSH" title="MedGen record for Class I obesity">Class I obesity</a></span></li><li><span class="TLline"><a href="/medgen/281328" ref="tree=MeSH" title="MedGen record for Class II obesity">Class II obesity</a></span></li><li><span class="TLline"><a href="/medgen/284586" ref="tree=MeSH" title="MedGen record for Class III obesity">Class III obesity</a></span></li><li><span class="TLline"><a href="/medgen/18472" ref="tree=MeSH" title="MedGen record for Extreme obesity with alveolar hypoventilation">Extreme obesity with alveolar hypoventilation</a></span></li><li><span class="TLline"><a href="/medgen/888153" ref="tree=MeSH" title="MedGen record for Iatrogenic Obesity">Iatrogenic Obesity</a></span></li><li><span class="TLline"><a href="/medgen/885912" ref="tree=MeSH" title="MedGen record for Inherited obesity">Inherited obesity</a></span><ul><li><span class="TLline"><a href="/medgen/1814458" ref="tree=MeSH" title="MedGen record for Hereditary non-syndromic obesity">Hereditary non-syndromic obesity</a></span></li></ul></li><li><span class="TLline"><a href="/medgen/18128" ref="tree=MeSH" title="MedGen record for Morbid obesity">Morbid obesity</a></span></li><li><span class="TLline"><a href="/medgen/473282" ref="tree=MeSH" title="MedGen record for Obesity in Pregnancy">Obesity in Pregnancy</a></span></li><li><span class="TLline"><a href="/medgen/875725" ref="tree=MeSH" title="MedGen record for Obesity, Metabolically Benign">Obesity, Metabolically Benign</a></span></li><li><span class="TLline"><a href="/medgen/882417" ref="tree=MeSH" title="MedGen record for Polygenic Obesity">Polygenic Obesity</a></span></li><li><span class="TLline"><a href="/medgen/46057" ref="tree=MeSH" title="MedGen record for Prader-Willi syndrome">Prader-Willi syndrome</a></span><ul><li><span class="TLline"><a href="/medgen/1826086" ref="tree=MeSH" title="MedGen record for Prader-Willi syndrome due to imprinting mutation">Prader-Willi syndrome due to imprinting mutation</a></span></li><li><span class="TLline"><a href="/medgen/1826079" ref="tree=MeSH" title="MedGen record for Prader-Willi syndrome due to maternal uniparental disomy of chromosome 15">Prader-Willi syndrome due to maternal uniparental disomy of chromosome 15</a></span></li><li><span class="TLline"><a href="/medgen/1826129" ref="tree=MeSH" title="MedGen record for Prader-Willi syndrome due to paternal 15q11q13 deletion">Prader-Willi syndrome due to paternal 15q11q13 deletion</a></span></li><li><span class="TLline"><a href="/medgen/1826085" ref="tree=MeSH" title="MedGen record for Prader-Willi syndrome due to translocation">Prader-Willi syndrome due to translocation</a></span></li></ul></li><li><span class="TLline"><a href="/medgen/1637490" ref="tree=MeSH" title="MedGen record for Truncal obesity">Truncal obesity</a></span><ul><li><span class="TLline"><a href="/medgen/348475" ref="tree=MeSH" title="MedGen record for Childhood-onset truncal obesity">Childhood-onset truncal obesity</a></span></li></ul></li></ul></li><li><span class="TLline"><a href="/medgen/1625342" ref="tree=MeSH" title="MedGen record for Overweight in childhood">Overweight in childhood</a></span></li></ul></li></ul></li></ul></li></ul></li></ul></li></ul></div></div></div></div>
|
||
</div>
|
||
|
||
<div class="portlet mgSection" id="ID_112">
|
||
<div class="portlet_head mgSectionHead ui-widget-header"><h1 class="nl" id="Conditions_with_this_feature">Conditions with this feature</h1><a sid="112" href="#" class="portlet_shutter" title="Show/hide content"></a></div>
|
||
<div class="portlet_content ln clinfeat">
|
||
<div class="divPopper rprt" id="rdis_208639"><div><strong>Kleefstra syndrome 1</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>208639</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information."><span class="highlight" style="background-color:">C0795833</span></a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
|
||
<div class="spaceAbove">Kleefstra syndrome is characterized by intellectual disability, autistic-like features, childhood hypotonia, and distinctive facial features. The majority of individuals function in the moderate-to-severe spectrum of intellectual disability although a few individuals have mild delay and total IQ within low-normal range. While most have severe expressive speech delay with little speech development, general language development is usually at a higher level, making nonverbal communication possible. A complex pattern of other findings can also be observed; these include heart defects, renal/urologic defects, genital defects in males, severe respiratory infections, epilepsy / febrile seizures, psychiatric disorders, and extreme apathy or catatonic-like features after puberty.</div>
|
||
<div class="spaceAbove nowrap">See: <a href="/medgen/208639">Condition Record</a></div></div>
|
||
<div class="divPopper rprt" id="rdis_354526"><div><strong>Familial partial lipodystrophy, Dunnigan type</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>354526</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C1720860</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
|
||
<div class="spaceAbove">Familial partial lipodystrophy (FPLD) is a metabolic disorder characterized by abnormal subcutaneous adipose tissue distribution beginning in late childhood or early adult life. Affected individuals gradually lose fat from the upper and lower extremities and the gluteal and truncal regions, resulting in a muscular appearance with prominent superficial veins. In some patients, adipose tissue accumulates on the face and neck, causing a double chin, fat neck, or cushingoid appearance. Metabolic abnormalities include insulin-resistant diabetes mellitus with acanthosis nigricans and hypertriglyceridemia; hirsutism and menstrual abnormalities occur infrequently. Familial partial lipodystrophy may also be referred to as lipoatrophic diabetes mellitus, but the essential feature is loss of subcutaneous fat (review by Garg, 2004). The disorder may be misdiagnosed as Cushing disease (see 219080) (Kobberling and Dunnigan, 1986; Garg, 2004). Genetic Heterogeneity of Familial Partial Lipodystrophy Familial partial lipodystrophy is a clinically and genetically heterogeneous disorder. Types 1 and 2 were originally described as clinical subtypes: type 1 (FPLD1; 608600), characterized by loss of subcutaneous fat confined to the limbs (Kobberling et al., 1975), and FPLD2, characterized by loss of subcutaneous fat from the limbs and trunk (Dunnigan et al., 1974; Kobberling and Dunnigan, 1986). No genetic basis for FPLD1 has yet been delineated. FPLD3 (604367) is caused by mutation in the PPARG gene (601487) on chromosome 3p25; FPLD4 (613877) is caused by mutation in the PLIN1 gene (170290) on chromosome 15q26; FPLD5 (615238) is caused by mutation in the CIDEC gene (612120) on chromosome 3p25; FPLD6 (615980) is caused by mutation in the LIPE gene (151750) on chromosome 19q13; FPLD7 (606721) is caused by mutation in the CAV1 gene (601047) on chromosome 7q31; FPLD8 (620679), caused by mutation in the ADRA2A gene (104210) on chromosome 10q25; and FPLD9 (620683), caused by mutation in the PLAAT3 gene (613867) on chromosome 11q12.</div>
|
||
<div class="spaceAbove nowrap">See: <a href="/medgen/354526">Condition Record</a></div></div>
|
||
<div class="divPopper rprt" id="rdis_461968"><div><strong>Maturity-onset diabetes of the young type 11</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>461968</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C3150618</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Maturity-onset diabetes of the young (MODY) is a group of several conditions characterized by abnormally high levels of blood glucose, also called blood sugar. These forms of diabetes typically begin before age 30, although they can occur later in life. In MODY, elevated blood glucose arises from reduced production of insulin, which is a hormone produced in the pancreas that helps regulate blood glucose levels. Specifically, insulin controls how much glucose (a type of sugar) is passed from the blood into cells, where it is used as an energy source.\n\nThe different types of MODY are distinguished by their genetic causes. The most common types are HNF1A-MODY (also known as MODY3), accounting for 50 to 70 percent of cases, and GCK-MODY (MODY2), accounting for 30 to 50 percent of cases. Less frequent types include HNF4A-MODY (MODY1) and renal cysts and diabetes (RCAD) syndrome (also known as HNF1B-MODY or MODY5), which each account for 5 to 10 percent of cases. At least ten other types have been identified, and these are very rare.\n\nHNF1A-MODY and HNF4A-MODY have similar signs and symptoms that develop slowly over time. Early signs and symptoms in these types are caused by high blood glucose and may include frequent urination (polyuria), excessive thirst (polydipsia), fatigue, blurred vision, weight loss, and recurrent skin infections. Over time uncontrolled high blood glucose can damage small blood vessels in the eyes and kidneys. Damage to the light-sensitive tissue at the back of the eye (the retina) causes a condition known as diabetic retinopathy that can lead to vision loss and eventual blindness. Kidney damage (diabetic nephropathy) can lead to kidney failure and end-stage renal disease (ESRD). While these two types of MODY are very similar, certain features are particular to each type. For example, babies with HNF4A-MODY tend to weigh more than average or have abnormally low blood glucose at birth, even though other signs of the condition do not occur until childhood or young adulthood. People with HNF1A-MODY have a higher-than-average risk of developing noncancerous (benign) liver tumors known as hepatocellular adenomas.\n\nRCAD is associated with a combination of diabetes and kidney or urinary tract abnormalities (unrelated to the elevated blood glucose), most commonly fluid-filled sacs (cysts) in the kidneys. However, the signs and symptoms are variable, even within families, and not everyone with RCAD has both features. Affected individuals may have other features unrelated to diabetes, such as abnormalities of the pancreas or liver or a form of arthritis called gout.\n\nGCK-MODY is a very mild type of the condition. People with this type have slightly elevated blood glucose levels, particularly in the morning before eating (fasting blood glucose). However, affected individuals often have no symptoms related to the disorder, and diabetes-related complications are extremely rare.</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/461968">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_462406"><div><strong>Hereditary spastic paraplegia 51</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>462406</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C3151056</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">AP-4-associated hereditary spastic paraplegia (AP-4-HSP) is a childhood-onset and complex form of hereditary spastic paraplegia. Spastic paraparesis is a universal feature in affected individuals. Manifestations typically begin before age one year, with infants presenting with hypotonia, mild postnatal microcephaly, and delayed developmental milestones. Seizures are common in early childhood, often starting as prolonged febrile seizures. As the disease progresses, older children have intellectual disability that is usually moderate to severe; most affected individuals communicate nonverbally. Neurobehavioral/psychiatric manifestations (e.g., impulsivity, hyperactivity, and inattention) are common. Hypotonia transitions to progressive lower-extremity weakness and spasticity, accompanied by pyramidal signs such as plantar extension, ankle clonus, and hyperreflexia. Although some children achieve independent ambulation, most eventually lose this ability and rely on mobility aids or wheelchairs. In adolescence or early adulthood, spasticity may affect the upper extremities in some individuals but is generally less severe and not significantly disabling. Complications in some individuals include contractures, foot deformities, and bladder and bowel dysfunction. Dysphagia may emerge in advanced stages of the disease.</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/462406">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_481368"><div><strong>Hereditary spastic paraplegia 47</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>481368</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C3279738</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">AP-4-associated hereditary spastic paraplegia (AP-4-HSP) is a childhood-onset and complex form of hereditary spastic paraplegia. Spastic paraparesis is a universal feature in affected individuals. Manifestations typically begin before age one year, with infants presenting with hypotonia, mild postnatal microcephaly, and delayed developmental milestones. Seizures are common in early childhood, often starting as prolonged febrile seizures. As the disease progresses, older children have intellectual disability that is usually moderate to severe; most affected individuals communicate nonverbally. Neurobehavioral/psychiatric manifestations (e.g., impulsivity, hyperactivity, and inattention) are common. Hypotonia transitions to progressive lower-extremity weakness and spasticity, accompanied by pyramidal signs such as plantar extension, ankle clonus, and hyperreflexia. Although some children achieve independent ambulation, most eventually lose this ability and rely on mobility aids or wheelchairs. In adolescence or early adulthood, spasticity may affect the upper extremities in some individuals but is generally less severe and not significantly disabling. Complications in some individuals include contractures, foot deformities, and bladder and bowel dysfunction. Dysphagia may emerge in advanced stages of the disease.</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/481368">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_763877"><div><strong>X-linked central congenital hypothyroidism with late-onset testicular enlargement</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>763877</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C3550963</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Central hypothyroidism and testicular enlargement (CHTE) is characterized by central hypothyroidism, delayed pubertal testosterone rise, adult macroorchidism, variable prolactin deficiency, and occasional transient partial GH deficiency. Some carrier females also exhibit central hypothyroidism and mild prolactin deficiency. Inter- and intrafamilial variability has been observed (summary by Joustra et al., 2016).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/763877">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_766268"><div><strong>Deafness-encephaloneuropathy-obesity-valvulopathy syndrome</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>766268</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C3553354</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">CoQ10 deficiency-2 (COQ10D2) is an autosomal recessive disorder characterized by sensorineural deafness, optic atrophy, mildly impaired intellectual development, muscular weakness, and cardiac involvement (summary by Nardecchia et al., 2021).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/766268">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_767287"><div><strong>Intellectual developmental disorder with autism and macrocephaly</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>767287</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C3554373</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">CHD8-related neurodevelopmental disorder with overgrowth (CHD8-NDD) is characterized by generalized overgrowth, developmental delay / intellectual disability (DD/ID), autism spectrum disorder (ASD), neuropsychiatric issues, neurologic problems, sleep disturbance, and gastrointestinal issues The most common findings are the development of macrocephaly (most often during infancy) and tall stature (most typically during puberty), which is often accompanied by ASD and/or DD/ID. Most, if not all, affected individuals have some degree of DD, most commonly speech and motor delays. When present, ID is most often in the mild-to-moderate range. Sleep disturbance is characterized by difficulty with both initiation (delayed sleep onset) and maintenance (frequent night awakenings) of sleep. The most common gastrointestinal issue is constipation with or without periods of diarrhea. Less common features are hypotonia (about 30% of affected individuals), seizures (10%-15%), dystonia (rare), and Chiari I malformation (rare).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/767287">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_863995"><div><strong>Motor developmental delay due to 14q32.2 paternally expressed gene defect</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>863995</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C4015558</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Temple syndrome is a short stature disorder of imprinting. The cardinal features are low birth weight, hypotonia and motor delay, feeding problems early in life, early puberty, and significantly reduced final height. Facial features include a broad forehead and short nose with a wide nasal tip, and the majority of patients have small hands and feet. However, many of the clinical features are nonspecific, making diagnosis difficult. In addition, isodisomy may uncover recessive disorders, which may influence the phenotype in maternal uniparental disomy of chromosome 14 (UPD14mat) cases (summary by Ioannides et al., 2014).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/863995">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1374358"><div><strong>Bardet-biedl syndrome 21</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1374358</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C4319932</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">BBS21 is an autosomal recessive ciliopathy characterized by obesity, postaxial polydactyly, retinal degeneration, and mild cognitive impairment (Heon et al., 2016; Khan et al., 2016). For a general phenotypic description and a discussion of genetic heterogeneity of Bardet-Biedl syndrome, see BBS1 (209900).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1374358">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1615839"><div><strong>Intellectual disability, autosomal dominant 52</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1615839</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C4540478</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Mental or Behavioral Dysfunction</dd></dl></div></div></div>
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||
<div class="spaceAbove nowrap">See: <a href="/medgen/1615839">Condition Record</a></div></div>
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||
<div class="divPopper rprt" id="rdis_1680544"><div><strong>Intellectual developmental disorder, X-linked 108</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1680544</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5193009</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">X-linked intellectual developmental disorder-108 (MRX108) is characterized by early hypotonia, global developmental delay, and moderately to severely impaired intellectual development. Brisk tendon reflexes, variable facial dysmorphism, and fifth finger clinodactyly may be present (Khayat et al., 2019).</div>
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||
<div class="spaceAbove nowrap">See: <a href="/medgen/1680544">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1782465"><div><strong>Myofibrillar myopathy 11</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1782465</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5543038</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Myofibrillar myopathy-11 (MFM11) is an autosomal recessive skeletal muscle disorder characterized by onset of slowly progressive proximal muscle weakness in the first decade of life. Some patients may present at birth with hypotonia and feeding difficulties, whereas others present later in mid-childhood. Although most patients show delayed walking at 2 to 3 years, all remain ambulatory into adulthood. More variable features may include decreased respiratory forced vital capacity, variable cardiac features, and calf hypertrophy. Skeletal muscle biopsy shows myopathic changes with variation in fiber size, type 1 fiber predominance, centralized nuclei, eccentrically placed core-like lesions, and distortion of the myofibrillary pattern with Z-line streaming and abnormal myofibrillar aggregates or inclusions (summary by Donkervoort et al., 2020). For a phenotypic description and a discussion of genetic heterogeneity of myofibrillar myopathy, see MFM1 (601419).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1782465">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1781649"><div><strong>Kohlschutter-Tonz syndrome-like</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1781649</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5543202</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Den Hoed-de Boer-Voisin syndrome (DHDBV) is characterized by global developmental delay with moderately to severely impaired intellectual development, poor or absent speech, and delayed motor skills. Although the severity of the disorder varies, many patients are nonverbal and have hypotonia with inability to sit or walk. Early-onset epilepsy is common and may be refractory to treatment, leading to epileptic encephalopathy and further interruption of developmental progress. Most patients have feeding difficulties with poor overall growth and dysmorphic facial features, as well as significant dental anomalies resembling amelogenesis imperfecta. The phenotype is reminiscent of Kohlschutter-Tonz syndrome (KTZS; 226750). More variable features of DHDBV include visual defects, behavioral abnormalities, and nonspecific involvement of other organ systems (summary by den Hoed et al., 2021).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1781649">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1794167"><div><strong>Developmental delay, impaired speech, and behavioral abnormalities</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1794167</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5561957</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Developmental delay, impaired speech, and behavioral abnormalities (DDISBA) is characterized by global developmental delay apparent from early childhood. Intellectual disability can range from mild to severe. Additional variable features may include dysmorphic facial features, seizures, hypotonia, motor abnormalities such as Tourette syndrome or dystonia, and hearing loss (summary by Cousin et al., 2021).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1794167">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1794210"><div><strong>Joubert syndrome 39</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1794210</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5562000</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Joubert syndrome-39 (JBTS39) is an autosomal recessive neurodevelopmental disorder with variable manifestations. Most affected individuals have developmental delay with poor speech and retinal dystrophy with abnormal eye movements. Brain imaging shows the pathognomonic 'molar tooth sign,' which reflects abnormal cerebellar formation (Van De Weghe et al., 2021). For a discussion of genetic heterogeneity of Joubert syndrome, see JBTS1 (213300).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1794210">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1794214"><div><strong>Developmental delay with or without intellectual impairment or behavioral abnormalities</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1794214</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5562004</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Developmental delay with or without intellectual impairment or behavioral abnormalities (DDIB) is an autosomal dominant disorder with a nonspecific phenotype of developmental delay. Additional features may include neonatal feeding problems, hypotonia, and dysmorphic facial features (Dulovic-Mahlow et al., 2019; van Woerden et al., 2021).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1794214">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1802903"><div><strong>Macrocephaly, neurodevelopmental delay, lymphoid hyperplasia, and persistent fetal hemoglobin</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1802903</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5676928</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Macrocephaly, neurodevelopmental delay, lymphoid hyperplasia, and persistent fetal hemoglobin (MNDLFH) is characterized by clinically significant pharyngeal lymphoid hypertrophy, with adenoid overgrowth, frequent upper airway infections, and sleep apnea. Macrocephaly without structural brain abnormalities is present, and patients exhibit increased weight for height as well as delayed gross motor and impaired intellectual development; autistic features and attention-deficit hyperactivity disorder have also been reported. An increased fraction of fetal hemoglobin has been observed in some patients (Ohishi et al., 2020; von der Lippe et al., 2022).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1802903">Condition Record</a></div></div>
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<div class="divPopper rprt" id="rdis_1823997"><div><strong>Developmental delay, behavioral abnormalities, and neuropsychiatric disorders</strong><div class="aux"><div class="resc"><dl class="rprtid"><dt>MedGen UID: </dt><dd>1823997</dd><dt><span class="dotprefix"> •</span>Concept ID: </dt><dd><a href="/medgen/docs/help/#sources" target="_blank" title="Concept Unique Identifier from NLM's Unified Medical Language system (UMLS) Click for more information.">C5774224</a></dd><dt><span class="dotprefix"> •</span></dt><dd>Disease or Syndrome</dd></dl></div></div></div>
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<div class="spaceAbove">Developmental delay, behavioral abnormalities, and neuropsychiatric disorders (DEDBANP) is a neurodevelopmental disorder characterized by mild global developmental delay and normal or variably impaired intellectual development. Most individuals have behavioral or neuropsychiatric disorders, including autism spectrum disorder (ASD), attention deficit-hyperactivity disorder (ADHD), and executive functioning deficits. Additional features may include speech delay, dysmorphic features, hypotonia, sleep disturbances, and seizures (Vitobello et al., 2022).</div>
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<div class="spaceAbove nowrap">See: <a href="/medgen/1823997">Condition Record</a></div></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1374358" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Bardet-biedl syndrome 21</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_766268" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Deafness-encephaloneuropathy-obesity-valvulopathy syndrome</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1794214" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Developmental delay with or without intellectual impairment or behavioral abnormalities</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1823997" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Developmental delay, behavioral abnormalities, and neuropsychiatric disorders</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1794167" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Developmental delay, impaired speech, and behavioral abnormalities</a></div><div class="jig-moreless" data-jigconfig="class: 'moveDown', moreText: 'See full list (19)', lessText: 'Show less', nodeBefore: 0"><span id="clinMore">
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_354526" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Familial partial lipodystrophy, Dunnigan type</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_481368" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Hereditary spastic paraplegia 47</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_462406" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Hereditary spastic paraplegia 51</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_767287" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Intellectual developmental disorder with autism and macrocephaly</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1680544" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Intellectual developmental disorder, X-linked 108</a></div>
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<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1615839" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Intellectual disability, autosomal dominant 52</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1794210" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Joubert syndrome 39</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_208639" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Kleefstra syndrome 1</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1781649" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Kohlschutter-Tonz syndrome-like</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1802903" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Macrocephaly, neurodevelopmental delay, lymphoid hyperplasia, and persistent fetal hemoglobin</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_461968" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Maturity-onset diabetes of the young type 11</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_863995" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Motor developmental delay due to 14q32.2 paternally expressed gene defect</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_1782465" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">Myofibrillar myopathy 11</a></div>
|
||
<div class="hangingIndent"><a title="click for more information" class="jig-ncbipopper" href="#rdis_763877" data-jigconfig="hasArrow: true, openEvent: 'click', closeEvent: 'mouseout', openAnimation: 'fadeIn', closeAnimation: 'fadeOut', triggerPosition: 'center right', destPosition: 'center left', arrowDirection: 'left'">X-linked central congenital hypothyroidism with late-onset testicular enlargement</a></div></span></div></div>
|
||
</div>
|
||
|
||
<div class="portlet mgSection" id="ID_105">
|
||
<div class="portlet_head mgSectionHead ui-widget-header"><h1 class="nl" id="Professional_guidelines">Professional guidelines</h1><a sid="105" href="#" class="portlet_shutter" title="Show/hide content"></a></div>
|
||
<div class="portlet_content ln"><h3 class="subhead">PubMed<a class="help jig-ncbi-popper" data-jig="ncbipopper" href="#guidelinesHelpPM"><img class="pulldown" src="//static.pubmed.gov/portal/portal3rc.fcgi/4223267/img/4204968" /></a></h3>
|
||
<div class="nl"><a target="_blank" href="/pubmed/37150579">American Association of Clinical Endocrinology Consensus Statement: Comprehensive Type 2 Diabetes Management Algorithm - 2023 Update.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Samson SL,
|
||
Vellanki P,
|
||
Blonde L,
|
||
Christofides EA,
|
||
Galindo RJ,
|
||
Hirsch IB,
|
||
Isaacs SD,
|
||
Izuora KE,
|
||
Low Wang CC,
|
||
Twining CL,
|
||
Umpierrez GE,
|
||
Valencia WM</span><br />
|
||
<span class="medgenPMjournal">Endocr Pract</span>
|
||
2023 May;29(5):305-340.
|
||
doi: 10.1016/j.eprac.2023.02.001.
|
||
<span class="bold">PMID: </span><a href="/pubmed/37150579" target="_blank">37150579</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/33246516">Best Practices in the Management of Overweight and Obesity.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Tchang BG,
|
||
Saunders KH,
|
||
Igel LI</span><br />
|
||
<span class="medgenPMjournal">Med Clin North Am</span>
|
||
2021 Jan;105(1):149-174.
|
||
Epub 2020 Nov 7
|
||
doi: 10.1016/j.mcna.2020.08.018.
|
||
<span class="bold">PMID: </span><a href="/pubmed/33246516" target="_blank">33246516</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/32278004">A new definition for metabolic dysfunction-associated fatty liver disease: An international expert consensus statement.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Eslam M,
|
||
Newsome PN,
|
||
Sarin SK,
|
||
Anstee QM,
|
||
Targher G,
|
||
Romero-Gomez M,
|
||
Zelber-Sagi S,
|
||
Wai-Sun Wong V,
|
||
Dufour JF,
|
||
Schattenberg JM,
|
||
Kawaguchi T,
|
||
Arrese M,
|
||
Valenti L,
|
||
Shiha G,
|
||
Tiribelli C,
|
||
Yki-Järvinen H,
|
||
Fan JG,
|
||
Grønbæk H,
|
||
Yilmaz Y,
|
||
Cortez-Pinto H,
|
||
Oliveira CP,
|
||
Bedossa P,
|
||
Adams LA,
|
||
Zheng MH,
|
||
Fouad Y,
|
||
Chan WK,
|
||
Mendez-Sanchez N,
|
||
Ahn SH,
|
||
Castera L,
|
||
Bugianesi E,
|
||
Ratziu V,
|
||
George J</span><br />
|
||
<span class="medgenPMjournal">J Hepatol</span>
|
||
2020 Jul;73(1):202-209.
|
||
Epub 2020 Apr 8
|
||
doi: 10.1016/j.jhep.2020.03.039.
|
||
<span class="bold">PMID: </span><a href="/pubmed/32278004" target="_blank">32278004</a></div>
|
||
<div><a target="_blank" href="https://pubmed.ncbi.nlm.nih.gov/?term=(%22overweight%22%5Btiab%3A~0%5D)%20AND%20(%22english%20and%20humans%22%5BFilter%5D)%20AND%20(%20(%22practice%20guideline%22%5BFilter%5D)%20OR%20(practice*%5Btitl%5D%20AND%20(guideline%5Btitl%5D%20OR%20parameter%5Btitl%5D%20OR%20resource%5Btitl%5D%20OR%20bulletin%5Btitl%5D%20OR%20best%5Btitl%5D))%20OR%20(genetic*%5Btitl%5D%20AND%20(evaluation%5Btitl%5D%20OR%20counseling%5Btitl%5D%20OR%20screening%5Btitl%5D%20OR%20test*%5Btitl%5D))%20OR%20(clinical%5Btitl%5D%20AND%20((expert%5Btitl%5D%20AND%20consensus%5Btitl%5D)%20OR%20utility%5Btitl%5D%20OR%20guideline*%5Btitl%5D))%20OR%20(management%5Btitl%5D%20AND%20(clinical%5Btitl%5D%20OR%20diagnos*%5Btitl%5D%20OR%20recommendation%5Btitl%5D%20OR%20pain%5Btitl%5D%20OR%20surveillance%5Btitl%5D%20OR%20emergency%5Btitl%5D%20OR%20guideline*%5Btitl%5D%20OR%20therap*))%20OR%20(treatment%5Btitl%5D%20AND%20((evaluation%5Btitl%5D%20AND%20diagnosis%5Btitl%5D)%20OR%20(assessment%5Btitl%5D%20AND%20prevention%5Btitl%5D)%20OR%20therap*))%20OR%20(Diagnos*%5Btitl%5D%20AND%20(prenatal%5Btitl%5D%20OR%20treatment%5Btitl%5D%20OR%20follow-up%5Btitl%5D%20OR%20statement%5Btitl%5D%20OR%20criteria%5Btitl%5D%20OR%20newborn%5Btitl%5D%20OR%20differential%5Btitl%5D%20OR%20neonatal%5Btitl%5D%20OR%20neonate%5Btitl%5D))%20OR%20(guideline*%5Btitl%5D%20AND%20(pharmacogenetic*%5Btitl%5D%20OR%20recommendation%5Btitl%5D%20OR%20therap*%5Btitl%5D%20OR%20evidence-based%5Btitl%5D%20OR%20consensus%5Btitl%5D%20OR%20(technical%5Btitl%5D%20AND%20standard*%5Btitl%5D)%20OR%20(molecular%5Btitl%5D%20AND%20testing%5Btitl%5D)))%20OR%20(risk%5Btitl%5D%20AND%20assessment%5Btitl%5D)%20OR%20(recommendation*%5Btitl%5D%20AND%20(statement%5Btitl%5D%20OR%20Evidence-based%5Btitl%5D%20OR%20Consensus%5Btitl%5D))%20OR%20(care%20AND%20((Patient%5Btitl%5D%20AND%20standard*%5Btitl%5D)%20OR%20primary%5Btitl%5D%20OR%20psychosocial%5Btitl%5D))%20OR%20(Health%5Btitl%5D%20AND%20supervision%5Btitl%5D)%20OR%20(statement%5Btitl%5D%20AND%20(policy%5Btitl%5D%20OR%20position%5Btitl%5D%20OR%20Consensus%5Btitl%5D))%20OR%20(pharmacogenetics%5Btitl%5D%20AND%20(Dosing%5Btitl%5D%20OR%20therap*%5Btitl%5D%20OR%20genotype*%5Btitl%5D%20OR%20drug*%5Btitl%5D))%20OR%20(Chemotherapy%5Btitl%5D%20AND%20decision*%5Btitl%5D)%20OR%20(screening%5Btitl%5D%20AND%20(newborn%5Btitl%5D%20OR%20neonat*%5Btitl%5D%20OR%20detection%5Btitl%5D%20OR%20diagnos*%5Btitl%5D))%20OR%20(criteria%5Btitl%5D%20OR%20genotype*%5Btitl%5D)%20)%20NOT%20(%22Case%20reports%22%5BPublication%20type%5D%20OR%20%22clinical%20study%22%5BPublication%20Type%5D%20OR%20%22randomized%20controlled%20trial%22%5BPublication%20Type%5D)" title="PubMed search">See all (3361)</a></div></div>
|
||
</div>
|
||
<div class="display-none help-popup" id="guidelinesHelpPM">These guidelines are articles in PubMed that match specific search criteria developed by MedGen to capture the most relevant practice guidelines. This list may not be comprehensive and may include broader topics as well. See the <a href="/medgen/docs/faq/" title="Frequently asked questions" target="_blank">FAQ</a> for details.</div><div class="display-none help-popup" id="guidelinesHelpCurated">These guidelines are manually curated by the MedGen team
|
||
to supplement articles available in PubMed. See the <a href="/medgen/docs/faq/" title="Frequently asked questions" target="_blank">FAQ</a> for details.</div>
|
||
<div class="portlet mgSection" id="ID_103">
|
||
<div class="portlet_head mgSectionHead ui-widget-header"><h1 class="nl" id="Recent_clinical_studies">Recent clinical studies</h1><a sid="103" href="#" class="portlet_shutter" title="Show/hide content"></a></div>
|
||
<div class="portlet_content ln"><h3 class="subhead">Etiology</h3>
|
||
<div class="nl"><a target="_blank" href="/pubmed/37775510">The global prevalence of overweight and obesity among nurses: A systematic review and meta-analyses.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Sadali UB,
|
||
Kamal KKBN,
|
||
Park J,
|
||
Chew HSJ,
|
||
Devi MK</span><br />
|
||
<span class="medgenPMjournal">J Clin Nurs</span>
|
||
2023 Dec;32(23-24):7934-7955.
|
||
Epub 2023 Sep 29
|
||
doi: 10.1111/jocn.16861.
|
||
<span class="bold">PMID: </span><a href="/pubmed/37775510" target="_blank">37775510</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/37594616">Upstream Determinants of Overweight and Obesity in Europe.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Vandevijvere S,
|
||
De Pauw R,
|
||
Djojosoeparto S,
|
||
Gorasso V,
|
||
Guariguata L,
|
||
Løvhaug AL,
|
||
Mialon M,
|
||
Van Dam I,
|
||
von Philipsborn P</span><br />
|
||
<span class="medgenPMjournal">Curr Obes Rep</span>
|
||
2023 Dec;12(4):417-428.
|
||
Epub 2023 Aug 18
|
||
doi: 10.1007/s13679-023-00524-1.
|
||
<span class="bold">PMID: </span><a href="/pubmed/37594616" target="_blank">37594616</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/33569831">Influences of exercise interventions on overweight and obesity in children and adolescents.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Lee J</span><br />
|
||
<span class="medgenPMjournal">Public Health Nurs</span>
|
||
2021 May;38(3):502-516.
|
||
Epub 2021 Feb 10
|
||
doi: 10.1111/phn.12862.
|
||
<span class="bold">PMID: </span><a href="/pubmed/33569831" target="_blank">33569831</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/33104108">Overweight: The Overlooked Risk Factor.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Kushner RF,
|
||
Primack C</span><br />
|
||
<span class="medgenPMjournal">J Fam Pract</span>
|
||
2020 Sep;69(7 Suppl):S51-S56.
|
||
<span class="bold">PMID: </span><a href="/pubmed/33104108" target="_blank">33104108</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/28404492">Overweight and obesity in Indonesia: prevalence and risk factors-a literature review.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Rachmi CN,
|
||
Li M,
|
||
Alison Baur L</span><br />
|
||
<span class="medgenPMjournal">Public Health</span>
|
||
2017 Jun;147:20-29.
|
||
Epub 2017 Mar 6
|
||
doi: 10.1016/j.puhe.2017.02.002.
|
||
<span class="bold">PMID: </span><a href="/pubmed/28404492" target="_blank">28404492</a></div>
|
||
<div><a target="_blank" href="https://pubmed.ncbi.nlm.nih.gov/?term=%22Overweight%22%20AND%20Etiology%2Fbroad%5Bfilter%5D%20%20AND%20%22english%20and%20humans%22%5Bfilter%5D%20NOT%20comment%5BPTYP%5D%20NOT%20letter%5BPTYP%5D" title="PubMed search">See all (59557)</a></div><h3 class="subhead">Diagnosis</h3>
|
||
<div class="nl"><a target="_blank" href="/pubmed/37277933">Mendelian randomization supports causality between overweight status and accelerated aging.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Chen Z,
|
||
Chen Z,
|
||
Jin X</span><br />
|
||
<span class="medgenPMjournal">Aging Cell</span>
|
||
2023 Aug;22(8):e13899.
|
||
Epub 2023 Jun 5
|
||
doi: 10.1111/acel.13899.
|
||
<span class="bold">PMID: </span><a href="/pubmed/37277933" target="_blank">37277933</a><a href="/pmc/articles/PMC10410004" target="_blank" class="PubMedFree">Free PMC Article</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/36050124">Nutrition recommendations for a healthy pregnancy and lactation in women with overweight and obesity - strategies for weight loss before and after pregnancy.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Hart TL,
|
||
Petersen KS,
|
||
Kris-Etherton PM</span><br />
|
||
<span class="medgenPMjournal">Fertil Steril</span>
|
||
2022 Sep;118(3):434-446.
|
||
Epub 2022 Aug 30
|
||
doi: 10.1016/j.fertnstert.2022.07.027.
|
||
<span class="bold">PMID: </span><a href="/pubmed/36050124" target="_blank">36050124</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/35183001">Overweight, obesity and assisted reproduction: A systematic review and meta-analysis.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Ribeiro LM,
|
||
Sasaki LMP,
|
||
Silva AA,
|
||
Souza ES,
|
||
Oliveira Lyrio A,
|
||
C M G Figueiredo A,
|
||
Gottems LBD</span><br />
|
||
<span class="medgenPMjournal">Eur J Obstet Gynecol Reprod Biol</span>
|
||
2022 Apr;271:117-127.
|
||
Epub 2022 Jan 31
|
||
doi: 10.1016/j.ejogrb.2022.01.019.
|
||
<span class="bold">PMID: </span><a href="/pubmed/35183001" target="_blank">35183001</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/31270831">Screen time and childhood overweight/obesity: A systematic review and meta-analysis.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Fang K,
|
||
Mu M,
|
||
Liu K,
|
||
He Y</span><br />
|
||
<span class="medgenPMjournal">Child Care Health Dev</span>
|
||
2019 Sep;45(5):744-753.
|
||
Epub 2019 Jul 24
|
||
doi: 10.1111/cch.12701.
|
||
<span class="bold">PMID: </span><a href="/pubmed/31270831" target="_blank">31270831</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/18607383">Global burden of obesity in 2005 and projections to 2030.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Kelly T,
|
||
Yang W,
|
||
Chen CS,
|
||
Reynolds K,
|
||
He J</span><br />
|
||
<span class="medgenPMjournal">Int J Obes (Lond)</span>
|
||
2008 Sep;32(9):1431-7.
|
||
Epub 2008 Jul 8
|
||
doi: 10.1038/ijo.2008.102.
|
||
<span class="bold">PMID: </span><a href="/pubmed/18607383" target="_blank">18607383</a></div>
|
||
<div><a target="_blank" href="https://pubmed.ncbi.nlm.nih.gov/?term=%22Overweight%22%20AND%20Diagnosis%2Fbroad%5Bfilter%5D%20%20AND%20%22english%20and%20humans%22%5Bfilter%5D%20NOT%20comment%5BPTYP%5D%20NOT%20letter%5BPTYP%5D" title="PubMed search">See all (18159)</a></div><h3 class="subhead">Therapy</h3>
|
||
<div class="nl"><a target="_blank" href="/pubmed/38819983">Tirzepatide for Weight Reduction in Chinese Adults With Obesity: The SURMOUNT-CN Randomized Clinical Trial.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Zhao L,
|
||
Cheng Z,
|
||
Lu Y,
|
||
Liu M,
|
||
Chen H,
|
||
Zhang M,
|
||
Wang R,
|
||
Yuan Y,
|
||
Li X</span><br />
|
||
<span class="medgenPMjournal">JAMA</span>
|
||
2024 Aug 20;332(7):551-560.
|
||
doi: 10.1001/jama.2024.9217.
|
||
<span class="bold">PMID: </span><a href="/pubmed/38819983" target="_blank">38819983</a><a href="/pmc/articles/PMC11337071" target="_blank" class="PubMedFree">Free PMC Article</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/37028708">Nutrition and Exercise Interventions to Improve Body Composition for Persons with Overweight or Obesity Near Retirement Age: A Systematic Review and Network Meta-Analysis of Randomized Controlled Trials.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Eglseer D,
|
||
Traxler M,
|
||
Embacher S,
|
||
Reiter L,
|
||
Schoufour JD,
|
||
Weijs PJM,
|
||
Voortman T,
|
||
Boirie Y,
|
||
Cruz-Jentoft A,
|
||
Bauer S;
|
||
SO-NUTS consortium</span><br />
|
||
<span class="medgenPMjournal">Adv Nutr</span>
|
||
2023 May;14(3):516-538.
|
||
Epub 2023 Apr 6
|
||
doi: 10.1016/j.advnut.2023.04.001.
|
||
<span class="bold">PMID: </span><a href="/pubmed/37028708" target="_blank">37028708</a><a href="/pmc/articles/PMC10201832" target="_blank" class="PubMedFree">Free PMC Article</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/32359762">Effects of l-carnitine supplementation on weight loss and body composition: A systematic review and meta-analysis of 37 randomized controlled clinical trials with dose-response analysis.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Talenezhad N,
|
||
Mohammadi M,
|
||
Ramezani-Jolfaie N,
|
||
Mozaffari-Khosravi H,
|
||
Salehi-Abargouei A</span><br />
|
||
<span class="medgenPMjournal">Clin Nutr ESPEN</span>
|
||
2020 Jun;37:9-23.
|
||
Epub 2020 Apr 18
|
||
doi: 10.1016/j.clnesp.2020.03.008.
|
||
<span class="bold">PMID: </span><a href="/pubmed/32359762" target="_blank">32359762</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/28923198">The epidemiology of sleep and obesity.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Ogilvie RP,
|
||
Patel SR</span><br />
|
||
<span class="medgenPMjournal">Sleep Health</span>
|
||
2017 Oct;3(5):383-388.
|
||
Epub 2017 Aug 15
|
||
doi: 10.1016/j.sleh.2017.07.013.
|
||
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<div class="nl"><a target="_blank" href="/pubmed/16034868">Interventions for preventing obesity in children.</a></div>
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<div class="nl"><a target="_blank" href="/pubmed/30389323">Association of BMI with overall and cause-specific mortality: a population-based cohort study of 3·6 million adults in the UK.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Bhaskaran K,
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Epub 2018 Oct 30
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<div class="nl"><a target="_blank" href="/pubmed/22274633">Obesity and burns.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Goutos I,
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Sadideen H,
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Pandya AA,
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<span class="bold">PMID: </span><a href="/pubmed/22274633" target="_blank">22274633</a></div>
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<div class="nl"><a target="_blank" href="/pubmed/19283243">Not mission impossible.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Parsons J</span><br />
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2009 Mar;38(3):85.
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<div class="nl"><a target="_blank" href="/pubmed/12513041">Obesity in adulthood and its consequences for life expectancy: a life-table analysis.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Peeters A,
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Barendregt JJ,
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Willekens F,
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Al Mamun A,
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2003 Jan 7;138(1):24-32.
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<div><a target="_blank" href="https://pubmed.ncbi.nlm.nih.gov/?term=%22Overweight%22%20AND%20Prognosis%2Fbroad%5Bfilter%5D%20%20AND%20%22english%20and%20humans%22%5Bfilter%5D%20NOT%20comment%5BPTYP%5D%20NOT%20letter%5BPTYP%5D" title="PubMed search">See all (18818)</a></div><h3 class="subhead">Clinical prediction guides</h3>
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<div class="nl"><a target="_blank" href="/pubmed/39045963">Accuracy of neck circumference in the diagnosis of overweight in children.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Traebert GA,
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2024;70(6):e20240049.
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<span class="bold">PMID: </span><a href="/pubmed/39045963" target="_blank">39045963</a><a href="/pmc/articles/PMC11288280" target="_blank" class="PubMedFree">Free PMC Article</a></div>
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<div class="nl"><a target="_blank" href="/pubmed/33238736">Role of gender, age and BMI in prognosis of heart failure.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Sciomer S,
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Moscucci F,
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Salvioni E,
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Marchese G,
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2020 Dec;27(2_suppl):46-51.
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doi: 10.1177/2047487320961980.
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<span class="bold">PMID: </span><a href="/pubmed/33238736" target="_blank">33238736</a><a href="/pmc/articles/PMC7691623" target="_blank" class="PubMedFree">Free PMC Article</a></div>
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<div class="nl"><a target="_blank" href="/pubmed/31778375">Taking BMI off the table.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Carey L</span><br />
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<span class="medgenPMjournal">N Z Med J</span>
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2019 Nov 8;132(1506):77-80.
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<div class="nl"><a target="_blank" href="/pubmed/28063802">Resolving the KgA1c paradox in the management of type 2 diabetes mellitus.</a></div>
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Kalra S,
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Mane A</span><br />
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<span class="medgenPMjournal">Diabetes Metab Syndr</span>
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2017 Nov;11 Suppl 1:S159-S168.
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<span class="bold">PMID: </span><a href="/pubmed/28063802" target="_blank">28063802</a></div>
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<div class="nl"><a target="_blank" href="/pubmed/12831945">Body composition interpretation. Contributions of the fat-free mass index and the body fat mass index.</a></div>
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<div class="portlet_content ln"><span class="medgenPMauthor">Kyle UG,
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<span class="bold">PMID: </span><a href="/pubmed/12831945" target="_blank">12831945</a></div>
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<div><a target="_blank" href="https://pubmed.ncbi.nlm.nih.gov/?term=%22Overweight%22%20AND%20Clinical%20prediction%20guides%2Fbroad%5Bfilter%5D%20%20AND%20%22english%20and%20humans%22%5Bfilter%5D%20NOT%20comment%5BPTYP%5D%20NOT%20letter%5BPTYP%5D" title="PubMed search">See all (31397)</a></div></div>
|
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|
||
|
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<div class="portlet mgSection" id="ID_104">
|
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<div class="portlet_head mgSectionHead ui-widget-header"><h1 class="nl" id="Recent_systematic_reviews">Recent systematic reviews</h1><a sid="104" href="#" class="portlet_shutter" title="Show/hide content"></a></div>
|
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<div class="portlet_content ln">
|
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<div class="nl"><a target="_blank" href="/pubmed/36400097">Global prevalence of non-alcoholic fatty liver disease and non-alcoholic steatohepatitis in the overweight and obese population: a systematic review and meta-analysis.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Quek J,
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Chan KE,
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Wong ZY,
|
||
Tan C,
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Tan B,
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||
Lim WH,
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||
Tan DJH,
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||
Tang ASP,
|
||
Tay P,
|
||
Xiao J,
|
||
Yong JN,
|
||
Zeng RW,
|
||
Chew NWS,
|
||
Nah B,
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||
Kulkarni A,
|
||
Siddiqui MS,
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||
Dan YY,
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||
Wong VW,
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||
Sanyal AJ,
|
||
Noureddin M,
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||
Muthiah M,
|
||
Ng CH</span><br />
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||
<span class="medgenPMjournal">Lancet Gastroenterol Hepatol</span>
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||
2023 Jan;8(1):20-30.
|
||
Epub 2022 Nov 16
|
||
doi: 10.1016/S2468-1253(22)00317-X.
|
||
<span class="bold">PMID: </span><a href="/pubmed/36400097" target="_blank">36400097</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/36012064">Ketogenic Diet Benefits to Weight Loss, Glycemic Control, and Lipid Profiles in Overweight Patients with Type 2 Diabetes Mellitus: A Meta-Analysis of Randomized Controlled Trails.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Zhou C,
|
||
Wang M,
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||
Liang J,
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||
He G,
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||
Chen N</span><br />
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||
<span class="medgenPMjournal">Int J Environ Res Public Health</span>
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||
2022 Aug 22;19(16)
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||
doi: 10.3390/ijerph191610429.
|
||
<span class="bold">PMID: </span><a href="/pubmed/36012064" target="_blank">36012064</a><a href="/pmc/articles/PMC9408028" target="_blank" class="PubMedFree">Free PMC Article</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/34426171">A systematic literature review on obesity: Understanding the causes & consequences of obesity and reviewing various machine learning approaches used to predict obesity.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Safaei M,
|
||
Sundararajan EA,
|
||
Driss M,
|
||
Boulila W,
|
||
Shapi'i A</span><br />
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||
<span class="medgenPMjournal">Comput Biol Med</span>
|
||
2021 Sep;136:104754.
|
||
Epub 2021 Aug 16
|
||
doi: 10.1016/j.compbiomed.2021.104754.
|
||
<span class="bold">PMID: </span><a href="/pubmed/34426171" target="_blank">34426171</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/30324651">Determinants of weight loss maintenance: a systematic review.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Varkevisser RDM,
|
||
van Stralen MM,
|
||
Kroeze W,
|
||
Ket JCF,
|
||
Steenhuis IHM</span><br />
|
||
<span class="medgenPMjournal">Obes Rev</span>
|
||
2019 Feb;20(2):171-211.
|
||
Epub 2018 Oct 16
|
||
doi: 10.1111/obr.12772.
|
||
<span class="bold">PMID: </span><a href="/pubmed/30324651" target="_blank">30324651</a><a href="/pmc/articles/PMC7416131" target="_blank" class="PubMedFree">Free PMC Article</a></div>
|
||
|
||
<div class="nl"><a target="_blank" href="/pubmed/20194822">Overweight, obesity, and depression: a systematic review and meta-analysis of longitudinal studies.</a></div>
|
||
<div class="portlet_content ln"><span class="medgenPMauthor">Luppino FS,
|
||
de Wit LM,
|
||
Bouvy PF,
|
||
Stijnen T,
|
||
Cuijpers P,
|
||
Penninx BW,
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Zitman FG</span><br />
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<span class="medgenPMjournal">Arch Gen Psychiatry</span>
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2010 Mar;67(3):220-9.
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<span class="bold">PMID: </span><a href="/pubmed/20194822" target="_blank">20194822</a></div>
|
||
<div><a target="_blank" href="https://pubmed.ncbi.nlm.nih.gov/?term=%22Overweight%22%20AND%20systematic%5Bsb%5D%20AND%20%22english%20and%20humans%22%5Bfilter%5D%20NOT%20comment%5BPTYP%5D%20NOT%20letter%5BPTYP%5D" title="PubMed search">See all (3046)</a></div></div>
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